Carbon monoxide (CO) released from its pharmacological donor prevents esophageal mucosa against experimental reflux esophagitis. Possible involvement of annexin 1, prostaglandin E2, endogenous hydrogen sulfide producing enzymes and selected vasoactive mechanisms

(1) Jagiellonian University Medical College,Cracow,Poland

(2) Jagiellonian University Collegium Medicum,Cracow,Poland

This item was part of the Oesophageal, Gastric and Duodenal Disorders I (Posters) session at UEG Week 2019

This item can be cited as: United European Gastroenterology Journal Volume 7 Issue 8 (Supplement), October 2019